It can therefore be deduced that burst swimming in aerobic performance could be enhanced by increased amounts of lipophilic fuels; dominancy of lactate dehydrogenase and glycolytic enzymes; high-level buffering capacity for the muscles; raised myosin ATPase activity, Ca++ATPase activity that is coupled to K+ ionic activities, and/or parvalbumin concentration whose function is activated transport of all in the white myotomal muscle cells.
On the other hand, swimming can be sustained with increased oxygen supply to the red oxidizing muscles which are influenced by the well structured circulatory system; increased metabolism of aerobic energy sources that lead to the generation of ATP by the red muscle; well loaded lipogenic stores whose catabolism demand a more aerated environment in the red muscle to produce energy; a high number of mitochondria that are contained on the red muscle for effective oxidative phosphorylation and thus acting as a powerhouse, or increased aerobic enzymatic reactions of ATP production that rely on catalysis by the presence heat and oxygen.
